CHO-KvLQT1 minK on QPatch


Sophion Bioscience


Hervor L Olsen, Morten Sunesen



The slowly activating, delayed rectifier K+ channel is among other activities important for regulating the repolarization phase of cardiac action potentials. The KvLQT1/minK channel consists of two transmembrane proteins. Mutations in the genes KCNQ1/KCNE1 coding for KvLQT1/minK are associated with predisposition to deafness, cardiac arrhythmia syndromes including long QT syndrome, atrial fibrillation and sudden infant death syndrome.

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